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We find that diet- and microbiota-dependent seizure protection is associated with elevations in bulk GABA relative to glutamate content in the hippocampus (Figure 6F)

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Using the framework of iron as a catalyst for lipid peroxidation, along with evidence of lipid peroxidation and decreased glutathione (the substrate for the ferroptosis checkpoint inhibitor, glutathione peroxidase 4, GPX4), ferroptosis has been implicated as a pathological contributor promoting neuronal loss [31,32]